The leaves fall, yet the tree endures.

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Publikace nespadá pod Ústav výpočetní techniky, ale pod Středoevropský technologický institut. Oficiální stránka publikace je na webu muni.cz.
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FOJTOVÁ Miloslava PROCHÁZKOVÁ SCHRUMPFOVÁ Petra FAJKUS Jiří

Rok publikování 2025
Druh Článek v odborném periodiku
Časopis / Zdroj CELLULAR AND MOLECULAR LIFE SCIENCES
Fakulta / Pracoviště MU

Středoevropský technologický institut

Citace
www https://link.springer.com/article/10.1007/s00018-025-05874-8
Doi https://doi.org/10.1007/s00018-025-05874-8
Klíčová slova Epigenetics; Meristem; Plant aging; Plant senescence;Telomere biology
Přiložené soubory
Popis Aging in plants presents a paradox: while individual modules such as leaves and reproductive organs undergo senescence, the plant as a whole may display extraordinary longevity, enabled by its modular architecture and perpetually active meristems. This review explores aging and senescence in plants by challenging commonly held assumptions and integrating emerging insights from telomere biology and epigenetic regulation. We critically examine the role of telomere length as a determinant of replicative lifespan, arguing that its importance is often overstated, particularly in the context of plant systems where telomerase activity persists in meristematic tissues. In contrast, the epigenetic landscape—including DNA methylation, histone modifications, chromatin remodeling, and non-coding RNAs—plays a dynamic and increasingly appreciated role in orchestrating senescence at cellular and organ levels. We synthesize current understanding of how these chromatin-level mechanisms interact with developmental cues and environmental stresses to regulate genome stability, transcriptional reprogramming, and longevity. By integrating chromosomal and epigenetic processes, this review provides a refined conceptual framework for understanding plant aging and highlights new opportunities to enhance resilience and lifespan in crops and long-lived species through targeted manipulation of telomere maintenance and epigenetic pathways.
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